An Epiphany--Receptors!!!

Lost his post. Found it again. Edit this later.
Eventually we do stumble into what seems correct.

Since, per last post, exercise or speed work fails to causecalcification to change the internal character of the new born fibril or their patterns within the bone lattice, does calcification do anything for the exercising horse? If we are talking about optimizing calcification by speed work, what are we talking about?

It suddenly occurred to me that in the racing animal calcificationwill over time fill in spaces and weak spots in bone that would be left vacant in the pasture horse. Even more importantly,calcification would keep those spaces filled!

How does this happen? Some hopefully solid guess work--

Exercise produces calcium receptors in areas where without the stimulation of pressure caused by movement those receptors would never form. Possibly this is a step by step process--one fibril gains a receptor and then the adjoining fibril does, and so on. Osteoblasts would then move in to the receptor areas and start forming new fibrils. We may think--if this is the process--how this might be particularly effective in the more porous trabecular bone (at ends of cannons and in interior--two types of bone--trabecular which is soft and cortical or hard bone)

In this conception the idea of optimizing calcification seems to depend on creation of "receptors" that form on the surface of the fibrils. I think I am on to something here! There are numerous receptors. Here is a list:

http://www.iuphar-db.org/DATABASE/FamilyMenuForward?familyId=12

I will avoid any study of "receptors" and note that I have zero idea whether the receptors I refer to a "calcium" receptors since the receptors referred to may have another name.

Nevertheless, I am thinking that something will cause calcification to move into weak areas of bone and keep these areas filled--as long as the animal is exercising.

Additionally, all this would explain the process of detraining in bone, which I'll get into. After your horse has been laid off for 6 months, what happens in its cannon bones? Another thing I've bumped into in this regard is parathyroid hormone that stimulates calcification. Get into this later also.

Training:

Thurs. 7/29: slow 3 miles riderless. Horse recovering from sore front hoofs.

Fri. 7/30: snappy 3 miles riderless 15-17 sec/f speed.

Sat. 7/31: riderless 2 x 1 mile 13.5-14 sec/f + warm up and warm down. High energy w/o. Was surprise that Rod sustained strength and enthusiasm through both miles. Might have a miler here. Very hard w/o causes us to cancel the tack work.